Abstract
Immunopathogenesis of myasthenia was discussed on the grounds of current experimental and clinical studies. Reported research findings indicate the autoimmunological character of the disease. They also suggest that neuromuscular transmission disorders found in this condition result from blacking the postsynaptic membrane receptor sites by blood antibodies directed against acetylcholine receptors. In the second part of the paper methods used presently in the treatment of myasthenia are described.